By Jim W. Burgess, Philip A. Sinclair, Christophe M. Chretien, Jonathon Boucher (auth.), Associate Professor Sukhinder Kaur Cheema (eds.)
About the Series:
Advances in Biochemistry in Heath and Disease provides state of the art discussions in state of the art biochemical learn, delivering fascinating advancements that influence healthcare and ailment learn. Volumes within the sequence specialize in cross-disciplinary biomedical learn and think about a number of issues in biochemistry, mobilephone biology, molecular biology, and biomedicine.
Biochemistry of Atherosclerosis
Sukhinder Kaur Cheema
Biochemistry of Atherosclerosis examines atherosclerosis in nice aspect, targeting the danger of atherosclerosis, and the biochemical pathways concerned. It presents a breadth of data in addition to new insights right into a number of issues in terms of atherosclerosis from major scientists world wide who're on the leading edge of atherosclerosis examine. Biochemistry of Atherosclerosis is key analyzing for biomedical and medical researchers.
- Hyperlipidaemia and Atherosclerosis
- Diabetes prompted Atherosclerosis
- Hypertension precipitated Atherosclerosis
- Homocysteine Metabolism and Atherosclerosis
- Role of the Immune process in Atherosclerosis
- Role of Infectious brokers in Atherogenesis
- Dietary administration of Aherosclerosis
About the Editor:
Sukhinder Kaur Cheema is presently affiliate Professor of Biochemistry and a CIHR (Canadian Institutes of future health examine) New Investigator on the Memorial collage of Newfoundland. knowledgeable in dietary biochemistry, lipid metabolism and heart problems, she is move appointed in college of medication on the Memorial college of Newfoundland.
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Extra resources for Biochemistry of Atherosclerosis
J Lipid Res 40: 1294–1303, 1999. Brundert M, Greten H, Rinninger F, Heeren J: Hepatic lipase mediates an increase in selective uptake of HDL-associated cholesteryl esters by cells in culture independent from SR-BI. J Lipid Res 44: 1020–1032, 2003. Connelly MA, Klein SM, Azhar S, Abumrad NA, Williams DL: Comparison of class B scavenger receptors, CD36 and scavenger receptor BI (SR-BI), shows that both receptors mediate high density lipoprotein-cholesteryl ester selective uptake but SR-BI exhibits a unique enhancement of cholesteryl ester uptake.
Transgenic mice  and rabbits  overexpressing the human LCAT gene have been reported to have a modest reduction in fasting TG levels. Gene-targeted mice deficient in Chapter 2. The Role of LCAT in Atherosclerosis 33 LCAT activities are associated with elevated TG in a gene dose-dependent manner [36, 37]. Collectively, these data are in support of a mechanistic link between LCAT deficiency and HTG. In humans, a partial reduction in postheparin lipase activity has been observed in association with a fasting HTG , other possible contributions have not been explored.
Arch Intern Med 159: 1893–1900, 1999. 111. Altmann SW, Davis HR Jr, Yao X, Laverty M, Compton DS, Zhu LJ, Crona JH, Caplen MA, Hoos LM, Tetzloff G, Priestley T, Burnett DA, Strader CD, Graziano MP: The identification of intestinal scavenger receptor class B, type I (SR-BI) by expression cloning and its role in cholesterol absorption. Biochim Biophys Acta 1580: 77–93, 2002. K. Cheema, Springer, New York, 2006 2 The Role of LCAT in Atherosclerosis DOMINIC S. NG Abstract Lecithin–cholesterol acyltransferase (LCAT) is one of the major modulators of plasma high-density lipoprotein cholesterol (HDL-C) and plays a central role in the reverse cholesterol transport (RCT) process.